Headache Group

Comment

Comment by Thomas Antony on September 3, 2009 at 10:43am

I am collecting abstracts of relevance in the following site. It is a lot easier. Check out: http://headachecollection.blogspot.com/

Comment by dr.v.k.agarwal on August 13, 2009 at 6:14pm

dr thomas
v informative collection by you.
all is well but if nothing works,we should try acupuncture as well ,that may be palliative or curative treatment esp. so in all such headaches where the cause is unknown

Comment by Thomas Antony on August 13, 2009 at 3:37pm

Occipital Nerve Blocks: When and What to Inject?
Occipital nerve block is an effective treatment for cervicogenic headache, cluster headache, and occipital neuralgia. While a double blinded randomized placebo controlled clinical trial is lacking, multiple open label studies reported favorable results for migraine. Two other possible uses of ONB worthy of further study are use as a rescue treatment and as an adjunctive treatment for medication overuse headache. ONB may be effective for tension headache, but only under very specific circumstances. ONB is either ineffective or only effective under as yet unstudied circumstances for hemicrania continua and chronic paroxysmal hemicrania. Some practitioners use occipital nerve (ON) tenderness to palpation (TTP) or reproduction of headache pain with ON pressure (RHPONP) as selection criteria for identifying appropriate patients. While only a clinical trial can produce a definitive answer, current evidence suggests that these selection criteria are not necessary for cervicogenic headache or cluster headache. Occipital neuralgia by definition involves TTP of the ONs. Whether RHPONP or ON TTP predicts success in migraine is unclear, and may relate to whether steroids are used. A single blinded randomized controlled trial evaluating local anesthetic with steroids vs local anesthetic alone for transformed migraine reported slightly worse results with steroids, but there are several alternate explanations for this finding other than steroids being counterproductive. The technique of repetitive ONBs deserves further study.
CephalagiaJoshua Tobin, MD; Stephen Flitman, MD; Aug 2009

Comment by Thomas Antony on August 13, 2009 at 3:35pm

Methylprednisolone therapy for short-term prevention of SUNCT syndrome
Short-lasting unilateral neuralgiform headache attacks with conjunctival injection and tearing (SUNCT) is characterized by severe and frequent daily pain attacks causing transient physical disability for the patients during the headache period. Currently there is no option for abortive treatment of the attacks, mainly due to the short-lived nature and frequency of the repeated headaches, while highly efficacious therapy is also unavailable for short-term prevention. We report rapidly suppressed headache attacks with orally administered methylprednisolone in eight headache periods of three patients with idiopathic, episodic SUNCT syndrome. The remission was maintained until the period was over in all cases. Although the mechanism of methylprednisolone action is unclear, it is probably based on the anti-inflammatory effects of the drug.
Cephalalgia; A Trauninger, B Alkonyi, N Kovács, S Komoly & Z Pfund

Comment by Thomas Antony on June 11, 2009 at 1:19pm

Chronic rhinosinusitis gives a ninefold increased risk of chronic headache. The Akershus study of chronic headache

Cephalalgia

http://www3.interscience.wiley.com/journal/122421078/abstract?CRETRY=1&SRETRY=0

Comment by Thomas Antony on May 7, 2009 at 2:55pm

And now, a new type of headache, for sp'a'cial people ;-)


'Space Motion Sickness' (SMS) have been reported in astronauts earlier, and a headache they experienced in space used to be attributed to the same. Now researchers from Leiden University Medical Centre, Netherlands suggest a new terminology - SPACE HEADACHE - for the same, after studying 17 atronauts. It was not associated with SMS. They propose 'space headache' be classifed as a separate entity among secondary headaches attributed to homeostasis disorders.
--------------------------
See, BRIEF REPORT
Space headache: a new secondary headache
AA Vein 1 , H Koppen 2 , J Haan 1,3 , GM Terwindt 1 & MD Ferrari 1
1 Department of Neurology, Leiden University Medical Centre, Leiden, 2
Haga Hospital, The Haque, and 3 Rijnland Hospital, Leiderdorp, the
Netherlands
Correspondence to Alla A Vein MD, PhD, Department of Neurology, Leiden
University Medical Centre, PO Box 9600, 2300 RC Leiden, the Netherlands.
E-mail: a.a.vein@lumc.nl
Cephalalgia, Volume 29 Issue 6, Pages 683 - 686
Published Online: 23 Jan 2009
Copyright © 2009 International Headache Society

Comment by Thomas Antony on April 23, 2009 at 4:57pm

Hi friends,

If interested do this:-

Activate your free 3 month trial to Wiley-Blackwell Medical Journals and receive full-text access

Visit : http://www.interscience.wiley.com/medicine/freetrial

Comment by Thomas Antony on April 23, 2009 at 4:41pm

Peripheral mechanisms of pain and analgesia
(This is the abstract of the article in Brain Research Reviews, Vol 60, Issue 1; April 2009 pp 90-113)
"This review summarizes recent findings on peripheral mechanisms underlying the generation and inhibition of pain. The focus is on events occurring in peripheral injured tissues that lead to the sensitization and excitation of primary afferent neurons, and on the modulation of such mechanisms. Primary afferent neurons are of particular interest from a therapeutic perspective because they are the initial generator of noxious impulses traveling towards relay stations in the spinal cord and the brain. Thus, if one finds ways to inhibit the sensitization and/or excitation of peripheral sensory neurons, subsequent central events such as wind-up, sensitization and plasticity may be prevented. Most importantly, if agents are found that selectively modulate primary afferent function and do not cross the blood–brain-barrier, centrally mediated untoward side effects of conventional analgesics (e.g. opioids, anticonvulsants) may be avoided. This article begins with the peripheral actions of opioids, turns to a discussion of the effects of adrenergic co-adjuvants, and then moves on to a discussion of pro-inflammatory mechanisms focusing on TRP channels and nerve growth factor, their signaling pathways and arising therapeutic perspectives."

By Christoph Stein, J.David Clark, Uhtaek Oh, Michael R Vasko, George L.Wilcox, Aaron C.Overland, Todd W.Vanderah & RObert H.Spencer

Comment by dr.v.k.agarwal on April 17, 2009 at 12:00pm

v nice info dr thomas
thanks

Comment by Thomas Antony on April 17, 2009 at 10:32am

Headache, Visual Disturbances & Papilloedema

Idiopathic intracranial hypertension (IIH) is defined as increased intracranial pressure in the absence of intracranial mass or obstructive hydrocephalus. Over 80% of patients are overweight women. IIH is usually encountered in the neurology and ophthalmology practise as headaches, visual disturbance and papilloedema are the characteristic features of this syndrome. Patients with IIH also experience tinnitus, hearing loss, balance disturbance, cerebrospinal fluid (CSF) otorrhoea or rhinorrhoea and in some cases these otorhinological symptoms can be presenting features of this syndrome. IIH is also associated with obstructive sleep apnoea. Otolaryngologists should be familiar with this important condition as it can manifest a variety of symptoms that are more frequently seen in their clinics. Sometimes otolaryngologists may be involved in the surgical management of this condition, such as repair of CSF rhinorrhoea or otorrhoea or endoscopic optic nerve decompression. The aim of this review article is to familiarise the otolaryngologists with the important features of this unusual syndrome which may remain unrecognised in the otolaryngology practice.


Idiopathic intracranial hypertension in otolaryngology; Jindal M, Hiam L, Raman A, Rejali D; European Archives of Oto-Rhino-Laryngology (Apr 2009)

• ‹ Previous
• 1
• 2
• 3
• 4
• Next ›
• Page